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Top of pagePATHOGENIC MECHANISMS AND MOLECULAR BASISFactor V Leiden is a genetic disorder seen as an a poor anticoagulant response to APC. APC is an organic anticoagulant protein that cleaves and inactivates procoagulant Factors Va and VIIIa, Thereby downregulating further thrombin generation.5,6 APC inactivates Factor Va by cleavage at three different amino acid positions: R (Arginine) 306, R 506, as R 679. The term V Leiden refers back to the specific guanine to adenine substitution at nucleotide 1691 in the Factor V gene, Which predicts the alternative of glutamine for arginine at the Arg 506 APC cleavage site. in this single amino acid substitution, Factor Va is resistant to APC and is inactivated at a 10 fold slower rate than normal, generating increased thrombin generation. Factor V cleaved at position 506 also actions as a cofactor (and additionally Protein S) For APC mediated inactivation of part VIIIa. Loss of this anticoagulant activity of Factor V may induce thrombin generation. The occurrance varies by population (game 1). Heterozygosity for Factor V Leiden occurs in 3 of the US and European populations. The highest heterozygosity rate can be found in nfl bedding set queen Europe; The mutation is extremely rare in Asian, photography equipment, And indigenous Australian populations. inside of of Europe, The prevalence varies from 10 to 15% in southern Sweden and Greece to 2 in Italy and Spain.10 The mutation is situated in 3.8% of folks in France, But how often ranges from 1.3% in southwestern regions to 7.1% in northeastern portugal.9 in the, The prevalence reflects the world service of the mutation.11 It is within:
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