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Top of pageAbstractPrevious observations of the anatomical distribution and pharmacological profile of the dopamine D3 receptor (DRD3) Have indicated its near future role in antipsychotic drug action. Risperidone, highly effective first line atypical antipsychotic agent, Exhibits a somewhat high affinity for this receptor. recent surveys have reported an association of the Ser9Gly polymorphism in the DRD3 gene with therapeutic response to risperidone, But the outcome was inconsistent. We therefore postulated that the Ser9Gly polymorphism might be in linkage disequilibrium with an not noticed variant that exerts a direct influence on risperidone efficacy. the latest study genotyped eight single nucleotide polymorphisms (SNPs) Distributed in the DRD3 gene and examined five of these for association with treatment outcome, Following an 8 week time frame risperidone monotherapy in 130 schizophrenic patients from mainland China. Clinical symptoms were assessed before and after the treatment period, making use of Brief Psychiatry Rating Scale (BPRS). The confounding effects of non genetics were estimated and the baseline symptom score was included as a covariate for adjustment. Neither was any association observed between the five polymorphisms and repair in total BPRS scores nor was any combined effect of these variants detected in the haplotype analysis. Given the tight correlation between the clinical potency and the D2 binding affinity of the normal neuroleptics (Creese et al, 1976; Seeman et al, 1976), The dopamine D2 receptor is assumed to function as a principal target for antipsychotic action. In vivo imaging studies have further demonstrated the significance of dopamine D2 receptor occupancy in predicting antipsychotic response and side effects (Remington in addition,yet Kapur, 1999). The advent of novel antipsychotics and the personality of additional dopamine receptor subtypes, notwithstanding, Have allowed for new insights into the wider diversity of pharmacological actions of antipsychotic drugs.